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It is accepted by some that peri-implantmucositis is the precursor of peri-implantitis as it is accepted that gingivitis is theprecursor of periodontitis.
EPA receives a sub-mission that includes a full set of required studies.
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For instance, it was recently shown thatPKA-induced ICER, which was thought to be the main inhibitor of not only responding T celllymphokine secretion, but also proliferation is not essential for those functions because micethat had this gene removed developed normal responding T cell inhibitory Tregs that had anincreased expression of EPAC and lower that usual levels of PDEs (phosphodiesterases),which was likely due to the need for higher levels of cAMP because of the lower affinity forthe EPAC CBD (Vang 2013).
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